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ATXN2 Gene Defect Model Shows Promise for Neurodegenerative Disease Treatment
Russian scientists modeled an ATXN2 gene defect causing neurodegenerative diseases; abnormal CAG repeats disrupt protein synthesis, impacting nerve impulse transmission and correlating with disease onset age; future research will test drugs on mice to increase nuclear fluid viscosity.
ATXN2 Gene Defect Model Shows Promise for Neurodegenerative Disease Treatment
Russian scientists modeled an ATXN2 gene defect causing neurodegenerative diseases; abnormal CAG repeats disrupt protein synthesis, impacting nerve impulse transmission and correlating with disease onset age; future research will test drugs on mice to increase nuclear fluid viscosity.
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