Tag #Neurodegenerative Disease

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lemonde.fr

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Jan 28, 13:10

French Weather Presenter Catherine Laborde Dies at 73

Former TF1 weather presenter Catherine Laborde died on January 28th at 73 from Lewy body dementia, a disease she publicly discussed; her nearly 30-year career at TF1 and her open sharing of her experience with the illness leaves a lasting legacy.

French Weather Presenter Catherine Laborde Dies at 73

Former TF1 weather presenter Catherine Laborde died on January 28th at 73 from Lewy body dementia, a disease she publicly discussed; her nearly 30-year career at TF1 and her open sharing of her experience with the illness leaves a lasting legacy.

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How did Catherine Laborde's public and private life intersect, shaping her legacy beyond her weather presenting career?

Laborde's death highlights the impact of Lewy body dementia, a neurodegenerative disease affecting over 200,000 in France. Her open discussion of her illness, including in her book "Trembler," and her husband's later accounts of her condition, raised public awareness of this lesser-known disease. Her career at TF1 spanned almost three decades, showcasing her lasting influence on French television.

arabic.euronews.com

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Jan 19, 22:06

Huntington's Disease Onset Explained by Accelerating Genetic Mutation

New research reveals that the Huntington's disease-causing genetic mutation, involving CAG repeat expansion in the huntingtin gene, remains dormant until it surpasses 150 repeats, triggering toxic protein production and neuronal cell death; this process accelerates significantly after 80 repeats, ex...

Huntington's Disease Onset Explained by Accelerating Genetic Mutation

New research reveals that the Huntington's disease-causing genetic mutation, involving CAG repeat expansion in the huntingtin gene, remains dormant until it surpasses 150 repeats, triggering toxic protein production and neuronal cell death; this process accelerates significantly after 80 repeats, ex...

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What is the mechanism identified by the new Huntington's disease research, and how does it explain the disease's delayed onset?

Huntington's disease, caused by a genetic mutation, has puzzled scientists due to its late-onset symptoms despite the mutation being present from birth. New research reveals that the mutation remains harmless for decades, gradually increasing until it surpasses a threshold of 150 CAG repeats, triggering toxic protein production and neuronal cell death.

dailymail.co.uk

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Jan 16, 01:07

Xenon Gas Shows Promise as Alzheimer's Treatment in Mouse Study

A US study found inhaling xenon gas reduced brain inflammation and improved cognitive function in mice with Alzheimer's, leading to plans for a human trial; xenon's ability to cross the blood-brain barrier is key.

Xenon Gas Shows Promise as Alzheimer's Treatment in Mouse Study

A US study found inhaling xenon gas reduced brain inflammation and improved cognitive function in mice with Alzheimer's, leading to plans for a human trial; xenon's ability to cross the blood-brain barrier is key.

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What are the immediate implications of the study's findings on the treatment of Alzheimer's disease?

Inhaling xenon gas showed promising results in reducing brain inflammation and improving cognitive function in mice with Alzheimer's-like pathology, suggesting a potential new treatment avenue. A human trial is planned to assess safety and efficacy. The gas's ability to cross the blood-brain barrier is a key advantage.

pt.euronews.com

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Jan 23, 01:10

Rare Gene Mutation Linked to Late-Onset Alzheimer's

A study of an Italian family with six members affected by late-onset Alzheimer's disease reveals a rare mutation in the GRIN2C gene as a probable cause, offering a significant advancement in understanding the disease's genetic basis.

Rare Gene Mutation Linked to Late-Onset Alzheimer's

A study of an Italian family with six members affected by late-onset Alzheimer's disease reveals a rare mutation in the GRIN2C gene as a probable cause, offering a significant advancement in understanding the disease's genetic basis.

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What is the significance of the GRIN2C gene mutation discovery for Alzheimer's disease research and potential treatments?

A new study identified a rare mutation in the GRIN2C gene as a potential cause of late-onset Alzheimer's disease in an Italian family. Six out of 15 family members developed the disease, all carrying the mutation, while nine healthy members did not. This discovery offers a significant genetic clue for Alzheimer's research.

abcnews.go.com

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Jan 16, 19:09

Huntington's Disease: CAG Repeat Expansion Identified as Key Disease Mechanism

New research shows that the Huntington's disease mutation, involving a repeated DNA sequence (CAG), expands over time, remaining harmless until it surpasses 150 repeats, triggering toxic protein production and neuronal death; this discovery could lead to new treatment strategies.

Huntington's Disease: CAG Repeat Expansion Identified as Key Disease Mechanism

New research shows that the Huntington's disease mutation, involving a repeated DNA sequence (CAG), expands over time, remaining harmless until it surpasses 150 repeats, triggering toxic protein production and neuronal death; this discovery could lead to new treatment strategies.

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What is the key mechanism by which the Huntington's disease mutation causes neuronal damage, and what is its clinical significance?

Huntington's disease, a fatal hereditary disorder, is caused by a genetic mutation where a DNA sequence (CAG) repeats at least 40 times. This mutation remains harmless for decades but expands over time, eventually exceeding a threshold of about 150 repeats, leading to toxic protein production and neuronal death.

dailymail.co.uk

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Jan 8, 16:09

Head Injuries May Reactivate Brain Viruses, Triggering Alzheimer's: Lab Study

Laboratory research suggests that head injuries may reactivate dormant herpes viruses (like HSV-1) in the brain, triggering inflammation and potentially leading to Alzheimer's disease, due to beta-amyloid plaque buildup and tau protein formation; this was demonstrated in a model brain.

Head Injuries May Reactivate Brain Viruses, Triggering Alzheimer's: Lab Study

Laboratory research suggests that head injuries may reactivate dormant herpes viruses (like HSV-1) in the brain, triggering inflammation and potentially leading to Alzheimer's disease, due to beta-amyloid plaque buildup and tau protein formation; this was demonstrated in a model brain.

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How does head trauma potentially trigger Alzheimer's disease, according to this research?

Laboratory research suggests that head injuries may reactivate dormant herpes viruses in the brain, triggering inflammation and potentially leading to Alzheimer's disease. This reactivation causes beta-amyloid plaque buildup and tau protein formation, hallmarks of Alzheimer's. The study used a model brain to simulate the effects of head trauma.

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